Neuroscience: Changes in Nerve Cells Caused by Social Isolation May Contribute to the Development of Mental Illness

neurosciencestuff:

Reduced production of myelin, a type of protective nerve fiber that is lost in diseases like multiple sclerosis, may also play a role in the development of mental illness, according to researchers at the Graduate School of Biomedical Sciences at Mount Sinai School of Medicine. The study is…

"The moment we start to feel the way we think, we start to think the way we feel. The brains vicious circle. This vicious circle in repeat activates some genes and turns some other ones off. We start memorizing the way we feel as our personality. Our organism gets use to the level of chemical substances that circulates our bloodstream, surrounds our cells and inundates our brain. Most people believe that emotions are real, that emotions and feelings are the final product of our experiences. If there is no new experiences, or experiences lived differently, then we will always live on the actualization of past emotions. The same chemical reaction. Knowledge is what proceeds experience. Learning information is personalizing it and applying it. We must modify our behavior to create new experiences that also carry along new emotions. Any process of change requires unlearning and re-learning."
– From an interview in spanish to a neuroscientist called Joe Dispenza

Scientists Discover How the Brain Ages

Researchers at Newcastle University have revealed the mechanism by which neurons, the nerve cells in the brain and other parts of the body, age.

The aging process has its roots deep within the cells and molecules that make up our bodies. Experts have previously identified the molecular pathway that react to cell damage and stems the cell’s ability to divide, known as cell senescence.

However, in cells that do not have this ability to divide, such as neurons in the brain and elsewhere, little was understood of the aging process. Now a team of scientists at Newcastle University, led by Professor Thomas von Zglinicki have shown that these cells follow the same pathway.

Alzheimer's could be the most catastrophic impact of junk food There is evidence that poor diet is one cause of Alzheimer's. If ever there was a case for the precautionary principle, this is it

The association between Alzheimer’s and type 2 diabetes is long-established: type 2 sufferers are two to three times more likely to be struck by this form of dementia than the general population. There are also associations between Alzheimer’s and obesity and Alzheimer’s and metabolic syndrome (a complex of diet-related pathologies).

Researchers first proposed that Alzheimer’s was another form of diabetes in 2005. The authors of the original paper investigated the brains of 54 corpses, 28 of which belonged to people who had died of the disease. They found that the levels of both insulin and insulin-like growth factors in the brains of Alzheimer’s patients were much lower than those in the brains of people who had died of other causes. Levels were lowest in the parts of the brain most affected by the disease.

Their work led them to conclude that insulin and insulin-like growth factor are produced not only in the pancreas but also in the brain. Insulin in the brain has a host of functions: as well as glucose metabolism, it helps to regulate the transmission of signals from one nerve cell to another, and affects their growth, plasticity and survival.